Alzheimer's Disease

Alzheimer’s Disease

Alzheimer’s disease causes progressive cognitive decline due to the loss of neurons and neural connections. Stem cells may help by reducing inflammation, clearing amyloid plaques, and supporting the repair of neural tissue. CellMark360’s technology ensures your adipose-derived stem cells are optimally prepared to promote brain health, offering a potential avenue to slow cognitive decline and support memory and function in Alzheimer’s patients.

How Your Own Stem Cells Support Healing for Alzheimer’s Disease

Slows Cognitive Decline

SVF and ADSCs have shown the ability to stabilize or even improve cognitive scores in Alzheimer’s patients, helping extend quality of life.

Reduces Alzheimer’s Markers

Studies show reductions in key brain biomarkers like phosphorylated tau and β-amyloid, which are closely linked to the progression of Alzheimer’s.

Supports Brain Regeneration

Patients treated with SVF have shown increased hippocampal volume, a critical area for memory and learning, suggesting the potential for brain structure restoration.

Safe and Repeatable Delivery

ICV and IV infusions of autologous SVF/ADSCs have demonstrated high safety, with only mild, temporary side effects and the possibility for multiple treatment rounds.

Research confirms ADSCs reduce inflammation, repair cartilage, and improve function.

Molecular Biology Reports, 2019 – Duma et al. (SVF in Alzheimer’s Patients)

Study Summary:

31 patients with neurodegenerative disorders (10 with Alzheimer’s) received ICV injections of autologous SVF over 3 years using an Ommaya reservoir.

Results at 3 years:

80% of Alzheimer’s patients showed cognitive stability or improvement;
biomarker reductions in P-tau and β-amyloid;
one patient’s hippocampal volume improved from <5th to 48th percentile.

(Source: Duma et al.)

Alzheimer’s Research & Therapy, 2023 – Kim et al. (IV ADSCs in Mild Alzheimer’s)

Study Summary:

32 patients received intravenous autologous ADSCs. Compared to controls, the treatment group showed slower cognitive decline and biochemical improvements.

Results at 12 months:

Reduced P-tau, increased BDNF, and less decline in ADAS-Cog scores, supporting neuron protection and memory stability

(Source: Kim et al.)

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